In stress-sharing embryo, average stress reduced to zero at step 40 during sequential target morphogenesis; without-sharing stress settled at non-zero with <55 cells moving

Entities in the same semantic neighborhood but without a typed relation to this one — candidates for new edges or unrecognized duplicates.

• Stress-sharing embryos achieve perfect sequential target patterns; non-sharing limited to <1% improvement.finding0.894
  Stress-sharing embryos formed each sequential target with stress reducing to zero; non-sharing achieved only tiny improvements.
• Cells in stress-sharing embryo moved average Euclidean distance ~2500 units until gen 400 then ~2400; without-sharing moved ~200 units (30x30 grid)finding0.860
  Quantifies how stress sharing enables long-range cell movements.
• Sharing of stress between cells facilitates morphogenesis and increases robustness of morphogenetic outcomes.hypothesis0.838
  Prior hypothesis tested in the paper; from Levin 2022.
• Stress-sharing embryo utilized maximum competency of 4725 units until generation 400, then dropped to ~4500; without-sharing used ~100 unitsfinding0.836
  Links competency utilization to stress sharing and morphological success.
• Stress sharing populations reached target morphology by generation 400, significantly faster than hardwired (1000) or without-sharing populationsfinding0.834
• Radius of influence in embryo with stress sharing: 30 units at step 1 decreasing to 0 at step 85; without sharing: 5 at step 1 ceasing at step 10finding0.832
  Direct measurement of cognitive light cone enlargement by stress sharing.
• Stress sharing reduces the functional inertia of low-stress cells, increasing their plasticity and exploratory behavior.claim0.830
  Shared stress raises the 'temperature' or exploratory activity of neighboring cells, enabling passage for stressed cells.
• Stress-sharing increases radius of cellular influence: ~30 units avg (step 1) vs ~5 units (non-sharing); lasts 85 steps vs 10 steps.finding0.817