Stress sharing reduces the functional inertia of low-stress cells, increasing their plasticity and exploratory behavior.

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• Stress sharing works to increase cohesiveness of collectives by raising exploratory plasticity in neighboring cells, enabling long-distance cell influencehypothesis0.895
• We hypothesize that stress sharing improves morphogenetic efficiency and increases cohesiveness of multicellular collectives.hypothesis0.863
  Central predictive hypothesis motivating the quantitative agent-based modeling.
• Stress sharing improves morphogenetic efficiency of multicellular collectives.claim0.853
  Central interpretive claim supported by multiple findings.
• Sharing of stress between cells facilitates morphogenesis and increases robustness of morphogenetic outcomes.hypothesis0.850
  Prior hypothesis tested in the paper; from Levin 2022.
• Stress sharing is an easy way to achieve robustness in collectives made up of homeostatic subunits.claim0.849
  Generalization of the model's implications.
• By sharing stress with neighbors, one cell’s (or region’s) problem becomes everyone’s problem, leading to a higher willingness to adopt new configurations in which everyone’s free energy is lower (and thus, more optimal morphogenetic problem-solving).quote0.849
  Encapsulates the core idea of stress sharing as collective cooperation.
• Stress-sharing enables cell movement over longer distances (~2500 units vs ~200 units without sharing).finding0.842
  Stress-sharing cells moved average Euclidean distance of ~2500 units, vs ~200 units in non-sharing populations.
• By sharing stress with neighbors, one cell's (or region's) problem becomes everyone's problem, leading to a higher willingness to adopt new configurations in which everyone's free energy is lower.concept0.841