Stress-sharing as distributed coordination

Emergent collective intelligence through local biochemical signaling that increases cellular influence radius and enables faster morphogenesis without explicit altruism or centralized control.

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The papers/notes whose extracted claims & findings make up this cluster.

Stress sharing as cognitive glue for collective intelligences: A computational model of stress as a coordinator for morphogenesis6 members
Stress Sharing as cognitive glue for collective intelligences: A computational model of Dukkha as a coordinator for Morphogenesis6 members
cognitive-glue-and-alexander.md1 member

Bridges (2)

Other communities that share members with this one — cross-cutting threads or papers that sit at the seam between two themes.

Care as mechanism of intelligence13 shared
Stress-Care-Intelligence Loop Framework4 shared

Findings (8)

Stress sharing benefit scales with grid complexity (20x20, 30x30, 50x50) and becomes more pronounced in later evolutionary stages when mutations alone fail
Stress sharing increases cognitive light cone radius from ~5 to ~30 units, persisting through development.Cell radius of influence in stress-sharing embryos was ~30 units vs ~5 units without sharing; lasted through step 85 vs step 10.
Stress sharing populations reached target morphology by generation 400, significantly faster than hardwired (1000) or without-sharing populations
Stress-sharing advantage increases with problem complexity
Stress-sharing embryos achieve perfect sequential target patterns; non-sharing limited to <1% improvement.Stress-sharing embryos formed each sequential target with stress reducing to zero; non-sharing achieved only tiny improvements.
Stress-sharing enables cell movement over longer distances (~2500 units vs ~200 units without sharing).Stress-sharing cells moved average Euclidean distance of ~2500 units, vs ~200 units in non-sharing populations.
Stress-sharing increases radius of cellular influence: ~30 units avg (step 1) vs ~5 units (non-sharing); lasts 85 steps vs 10 steps.
Stress-sharing populations reach anatomical targets faster than hardwired or non-sharing populations.Populations with stress sharing discovered correct morphology by generation 500, vs non-sharing and hardwired (p≪0.01).

Claims (5)

Stress molecules leak from source cells and diffuse to neighbors, making shared stress a conserved signal for cooperation.Central hypothesis that stress sharing enables cooperation without explicit altruism by making individual problems collective.
Stress sharing is a simple, robust mechanism for enabling emergent collective problem-solving without explicit altruism.
Stress sharing reduces the functional inertia of low-stress cells, increasing their plasticity and exploratory behavior.Shared stress raises the 'temperature' or exploratory activity of neighboring cells, enabling passage for stressed cells.
Stress sharing serves as a 'cognitive glue' for collective intelligences, enabling cooperation without explicit altruism by de-localizing incentive to work toward group goals
Owner wiping and stress-sharing are anti-individuation: the Buddhist not-self reframed as shared cognitive load.