finding

active

finding:stress-sharing-populations-reach-anatomical-targets-faster-than-hardwired-or-non-sharing-populations

Stress-sharing populations reach anatomical targets faster than hardwired or non-sharing populations.

Populations with stress sharing discovered correct morphology by generation 500, vs non-sharing and hardwired (p≪0.01).

Source paper

extracted_from

Stress Sharing as cognitive glue for collective intelligences: A computational model of Dukkha as a coordinator for Morphogenesis

(2024) · Shreesha, Lakshwin · Levin, Michael

Neighborhood — ranked by edge-count

Claims (1)

claim

Stress molecules leak from source cells and diffuse to neighbors, making shared stress a conserved signal for cooperation.
supports
Central hypothesis that stress sharing enables cooperation without explicit altruism by making individual problems collective.

Communities (2)

community

Care as mechanism of intelligence
members_of
Care defined as stress-relief concern; proposed invariant linking biology, AI, and evolution.
Stress-sharing as distributed coordination
members_of
Emergent collective intelligence through local biochemical signaling that increases cellular influence radius and enables faster morphogenesis without explicit altruism or centralized control.

Related by similarity (8)

cosine ≥ 0.65 · no typed edge

Entities in the same semantic neighborhood but without a typed relation to this one — candidates for new edges or unrecognized duplicates.

Stress sharing populations reached target morphology by generation 400, significantly faster than hardwired (1000) or without-sharing populationsfinding0.897
Stress-sharing embryos achieve perfect sequential target patterns; non-sharing limited to <1% improvement.finding0.847
Stress-sharing embryos formed each sequential target with stress reducing to zero; non-sharing achieved only tiny improvements.
For 20x20 grid, stress-sharing population reached max phenotypic fitness by generation 100; hardwired reached max at generation 1000; without-sharing never reached maxfinding0.842
Demonstrates benefit of stress sharing across smaller grid complexity.
Stress sharing improves morphogenetic efficiency of multicellular collectives.claim0.836
Central interpretive claim supported by multiple findings.
Stress sharing will be favored by evolutionary processes.hypothesis0.824
Evolutionary fitness hypothesis tested in the GA.
Stress sharing is an easy way to achieve robustness in collectives made up of homeostatic subunits.claim0.822
Generalization of the model's implications.
We hypothesize that stress sharing improves morphogenetic efficiency and increases cohesiveness of multicellular collectives.hypothesis0.821
Central predictive hypothesis motivating the quantitative agent-based modeling.
Stress sharing reduces the functional inertia of low-stress cells, increasing their plasticity and exploratory behavior.claim0.817
Shared stress raises the 'temperature' or exploratory activity of neighboring cells, enabling passage for stressed cells.

Cross-corpus bridges (1)

same_concept_as · Nomic cosine

External markdown files that talk about the same concept as this entity.

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What molecular mechanisms implement stress-sharing across cellular populations?questions/what-molecular-mechanisms-implement-stresssharing-across-cellular-populations.md0.825