Stress sharing improves morphogenetic efficiency of multicellular collectives.

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• We hypothesize that stress sharing improves morphogenetic efficiency and increases cohesiveness of multicellular collectives.hypothesis0.948
  Central predictive hypothesis motivating the quantitative agent-based modeling.
• Stress sharing works to increase cohesiveness of collectives by raising exploratory plasticity in neighboring cells, enabling long-distance cell influencehypothesis0.881
• Stress sharing is an easy way to achieve robustness in collectives made up of homeostatic subunits.claim0.869
  Generalization of the model's implications.
• By sharing stress with neighbors, one cell’s (or region’s) problem becomes everyone’s problem, leading to a higher willingness to adopt new configurations in which everyone’s free energy is lower (and thus, more optimal morphogenetic problem-solving).quote0.866
  Encapsulates the core idea of stress sharing as collective cooperation.
• Stress sharing reduces the functional inertia of low-stress cells, increasing their plasticity and exploratory behavior.claim0.853
  Shared stress raises the 'temperature' or exploratory activity of neighboring cells, enabling passage for stressed cells.
• Stress sharing will be favored by evolutionary processes.hypothesis0.851
  Evolutionary fitness hypothesis tested in the GA.
• Stress-sharing populations reach anatomical targets faster than hardwired or non-sharing populations.finding0.836
  Populations with stress sharing discovered correct morphology by generation 500, vs non-sharing and hardwired (p≪0.01).
• Stress sharing serves as a 'cognitive glue' for collective intelligences, enabling cooperation without explicit altruism by de-localizing incentive to work toward group goalsclaim0.834