concept
active
concept:cancer-as-defection-from-multicellular-selfCancer as Defection from Multicellular Self
The model that cancer results from cells shrinking their computational boundaries (closing gap junctions) and reverting to unicellular goals.
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Claims (1)
claim
- Oncogene-induced gap junction closure shrinks the cognitive boundary of cells.
Related by similarity (8)
cosine ≥ 0.65 · no typed edgeEntities in the same semantic neighborhood but without a typed relation to this one — candidates for new edges or unrecognized duplicates.
- The view that cancer is a shrinking of the Self boundary due to gap junction closure, reverting cells to unicellular goals.
- Cancer is interpreted as cells reverting to unicellular selfishness due to loss of gap junctional coupling, shrinking their cognitive boundary.
- Cancer results from defection from large-scale anatomical goals due to reduction of gap junctional connectivity, causing cells to revert to unicellular concerns
- Cancer is a breakdown of the multiscale binding, leading cells to revert to unicellular goals.hypothesis0.805Gap junction closure makes cells selfish, shrink their cognitive boundary; restoring Vmem can normalize.
- Frames metastasis as cells reverting to unicellular goals, treatable by restoring bioelectric connectivity.
- Provocative reinterpretation of cancer as a failure mode of collective decision-making.
- Cancer understood as cells that shrink their self-model to a tiny radius and defect from the collective, treating the rest of the body as environment