Smooth muscle tension (especially latched) is involved in a wide range of health problems.

Entities in the same semantic neighborhood but without a typed relation to this one — candidates for new edges or unrecognized duplicates.

• Sum total of all latches likely accounts for the majority of bodily suffering.claim0.770
  Practical implication of latch-bridge mechanism.
• Latched Hyperprior Hypothesis (LHH): If a vascular contraction is held long enough, it engages the latch-bridge mechanism, durably freezing the nearby circuit and creating a durable commitment to a specific hyperprior isolated from global updating, unlocking only when the corresponding prediction is resolved.hypothesis0.742
  Third core hypothesis, explaining how latched VSMCs instantiate hyperpriors.
• "The world around us requires a special looseness to allow normal human life to run in its normal state."quote0.736
  A key claim that the environment must have a specific kind of looseness (living structure) to support ordinary human happiness.
• Cells should leak molecules such as HSP90 extracellularly as a stress sharing mechanism.claim0.736
  Prediction for in-vivo biology from the model.
• Stressed cells leak stress signals to neighbors, raising their plasticity and enabling them to create temporary passages (tunnels) for movement.hypothesis0.732
• Smooth muscle cell motifs are where the brain's top-down predictive models are hiding.claim0.731
  Key claim for the FEP-AI community about the physical location of predictive models.
• Stress, care, and intelligence manifest in continuous loops; successfully overcoming a stress factor through intelligence necessarily introduces new problem spaces.claim0.731
  Explaining the autogenerative nature of SCI loops.
• Stress-sharing populations reach anatomical targets faster than hardwired or non-sharing populations.finding0.728
  Populations with stress sharing discovered correct morphology by generation 500, vs non-sharing and hardwired (p≪0.01).