If cancer represents a shrinkage of the cognitive light cone, then interventions that restore cellular participation in tissue-level information-processing networks should suppress cancer progression.

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• Stress sharing will enlarge the effective cognitive light cone of individual cells.hypothesis0.819
  Testable hypothesis linking stress sharing to cognitive scale.
• Cancer can be seen as a breakdown of the binding process that scales Selves, with cells reverting to unicellular goals.claim0.818
  Oncogene-induced gap junction closure shrinks the cognitive boundary of cells.
• Cancer is a breakdown of the collective intelligence of tissues, a shrinking of the Self boundary due to gap junction closure.claim0.817
  Frames metastasis as cells reverting to unicellular goals, treatable by restoring bioelectric connectivity.
• The cancer phenotype can be reverted by artificially managing the bioelectric connections between a cell and its neighbors.hypothesis0.808
  Predicts that restoring gap junctional coupling or appropriate Vmem can normalize oncogene-expressing cells.
• Disruptions in cells' ability to join into a cohesive information-processing network via oncogenes and other factors correlate with cancer developmentfinding0.806
  Empirical support for the cancer-as-cognitive-defect / cognitive light cone shrinkage hypothesis
• Cancer can be normalized by reconnecting cells to the electrical network that harnesses them toward adaptive tissue homeostasis.claim0.805
  Proposes a non-toxic cancer treatment strategy.
• Stress sharing enlarges the cognitive light cone of individual cells.claim0.804
  Explains how stress sharing scales goal-directed activity.
• Cancer is a breakdown of the multiscale binding, leading cells to revert to unicellular goals.hypothesis0.792
  Gap junction closure makes cells selfish, shrink their cognitive boundary; restoring Vmem can normalize.